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NEW QUESTION: 1
The Internal Fault Map Class 1A(I1A)from the above print out is?

A. None of the above
B. I1A:21 and I1A:23
C. I1A:2 and I2A:4
D. I1A:6
E. I1A:18 and I1A:21
Answer: E

NEW QUESTION: 2
Your purchasing department wants to monitor overdue purchase orders. What are the prerequisites for issuing delivery reminders for a purchase order?
There are 3 correct answers to this question.
A. Output the purchase order.
B. Maintain purchasing value keys in the purchase order item.
C. Maintain a confirmation control key in the supplier master.
D. Set message determination in Customizing and maintain message records.
E. Maintain reminder periods in the purchase order item.
Answer: B,D,E

NEW QUESTION: 3
Select the class of Anti-diabetic medication that works in the specified organ to prevent hyperglycemia. Select all that applies. Brain (E)
A. Sulfonylureas
B. DPP4 Inhibitors
C. Biguanide
D. SGLT2 inhibitors
E. Alpha- Glucosidase Inhibitors
F. Thiazolidinediones
G. Glucagon-like peptide-1 receptor agonists
Answer: G
Explanation:
Explanation
Glucagon-like peptide-1 receptor agonists Sulfonylureas work in beta cells in the pancreas that are still functioning to enhance insulin secretion. Alpha-Glucosidase Inhibitors stop -glucosidase enzymes in the small intestine and delay digestion and absorption of starch and disaccharides which lowers the levels of glucose after meals. DPP4 blocks the degradation ofGLP-1, GIP, and a variety of other peptides, including brain natriuretic peptide. Glucagon-like peptide-1 receptor agonists work in various organs of the body.
Glucagon-like peptide-1 receptor agonists enhance glucose homeostasis through: (i) stimulation of insulin secretion; (ii) inhibition of glucagon secretion; (iii) direct and indirect suppression of endogenous glucose production; (iv) suppression of appetite; (v) enhanced insulin sensitivity secondary to weight loss; (vi) delayed gastric emptying, resulting in decreased postprandial hyperglycaemia. Thiazolidinediones are the only true insulin-sensitising agents, exerting their effects in skeletal and cardiac muscle, liver, and adipose tissue. It ameliorates insulin resistance, decreases visceral fat. Biguanides work in liver, muscle, adipose tissue via activation of AMP-activated protein kinase (AMPK) reduce hepatic glucose production. SGLT2 inhibitors work in the kidneys to inhibit sodium-glucose transport proteins to reabsorb glucose into the blood from muscle cells; overall this helps to improve insulin release from the beta cells of the pancreas.

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